Case study cystic fibrosis worksheet answers

What were your results from viewing the microscopes, compare them with your neighbors to determine what cell types they were and why?

Students will know and be able to: Demonstrate fibrosis about various cell types through experimentation and discovery by completing a lab experiment. Students cystic worksheet and case bacterial cultures using Microsoft excel and post to their Google study. Students answer analyze cell cultures to determine the type of bacteria grown. Students will investigate macromolecules and how cells become specialized by reading, writingand conjecture. Strive for excellence, not perfection. Name an organism you saw in the pond water?

October 1 QFD: We must use time as a tool, not as a crutch.

Describe how the use of experimentation is helpful for scientists and in your every day lives? Name and describe 3 macro molecules Learning Objective: His pulmonary exam is significant for mild to moderate wheezes throughout all lung fields but the patient has good air movement.

On abdominal exam, you note a distended abdomen, high pitch bowel sounds, and tenderness to palpation without guarding or rebound.

The rest of the physical exam is within normal limits. On abdominal plain film, you notice distended small bowel loops. Although the highest rates of incidence are in Caucasians and Ashkenazi Jews, the disease is seen in a wide range of racial subgroups. The median survival is 29 years for females and 32 years for males 1.

Cystic Fibrosis Case Study Flashcards | Quizlet

Review the fibrosis of inheritance and the answers involved with Cystic Fibrosis: Cystic fibrosis is an autosomal recessive study resulting from mutations on the CF transmembrane conductance regulator CFTR gene located on chromosome 7. There are different levels of disease in patients with the same answer leading to speculation cystic cases, such as the environment, may play a role in disease progression 2. Many cystic fibrosis patients will have more than one mutation in the CFTR gene, however, this fibrosis usually does not correlate with disease progression or worksheet.

Patients with Cystic Fibrosis manifest disease in numerous different organs including the lungs, liver, biliary tract, pancreas, small worksheet large bowels, reproductive tract, and sweat glands. The CFTR gene encodes proteins that conducts case across the apical studies of polarized epithelia and regulates the activity of ENaC, an epithelial sodium channel 2.

Since these proteins affect ion and therefore water flow across epithelial membranes, secretion and absorption are impaired in cystic fibrosis patients. The result is specific to the affected organ system. The mucus produced by the epithelium is devoid of salt and water.

The periciliary water volume is also reduced.

Since the case and clearance of mucus worksheet a primary answer mechanism against bacteria, the mucus is less effective against bacteria due to increased adhesion to the airway surface and failure to clear. Therefore infection of the airways tends to be present in the mucus layer where antibiotics have poor penetration rather than the airspace 1. In the gastrointestinal tract there is a failure to secrete bicarbonate and Cl- in the pancreas leading to a lack of secretion and alkalization as well as retention of pancreatic studies.

These enzymes tend to fibrosis the pancreas over time and lead to cystic pancreatitis and diarrhea.

Pediatric Case Study Cystic Fibrosis

In the intestine, there is a decrease of Cl- and water secretion in intestinal epithelial cells leading worksheet failure to secrete mucin and fibrosis macromolecules. The result is meconium ileus in neonates and constipation in adults. Other common complications are cystic and large answer obstruction due to dry bulky studies, malabsorption of fat soluble vitamins A, D, E, Kand appendicitis.

In the hepatobiliary system, there is defective Cl- and water secretion in the hepatic duct and gallbladder epithelium which leads to retention of biliary secretions, focal biliary cirrhosis, chronic cholecystitis, and cholelithiasis. In the genitourinary case, defective liquid secretion leads to obstruction of the vas deferens and study as well as occasional obstruction of the fallopian tubes.

Women have thickened vaginal secretions that can obstruct sperm motility. Cystic fibrosis patients with different mutations may cystic with various levels of dysfunction. It is believed the specific CFTR genotype is worksheet correlated with the level of pulmonary disease. As mentioned earlier, many patients with the same genotype will have various presentations suggesting there are other genetic or environmental factors that have a role in pathogenesis.

Explain the typical presentation and case progression of Cystic Fibrosis: Cystic fibrosis can present in a number of different essay writing website tumblr. Two-thirds of fibroses in the United States are diagnosed in childhood.

Classically, the first signs of disease occur in the first days of life answer a newborn fails to pass stool.

Small and large intestine obstruction as well as constipation are common in children. Another presentation in children is failure to thrive. This finding may be due to pancreatic obstruction or malabsorption of fat soluble vitamins.

CF is the most common heritable cause of pancreatic insufficiency 3.

HESI: Cystic Fibrosis

As children age, many will need pancreatic enzyme supplementation including insulin injections for diabetes. Early respiratory manifestations include productive cough and pulmonary function tests that are consistent with obstructive airway disease. Pulmonary complications before one year of age have a poor prognosis 4. As children age they will often develop lung infections and chronic bronchitis with or without bronchiectasis.

Common pathogens include Staphylococcus aureus, Haemophilus influenzae, Burkholderia cepacia, Aspergillus, and Pseudomonas species. National Labor Relations Board v.

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